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This study investigates the mechanism of toxicity of the pesticide ethylperfluorooctane sulfonamide (NEPFOS) and its metabolite perfluorooctane sulfonamide (PFOS) on isolated rabbit renal cortical mitochondria, finding that PFOS acts as a protonophore and uncouples oxidative phosphorylation, while NEPFOS requires metabolism to PFOS to exhibit cyt
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AR226-0240
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